BMD
at the University of South Alabama
Dr. Stanfield, Associate Professor, completed her B.S. and Ph.D. degrees in Physiology, with an emphasis on Neuroscience, at the University of California at Davis. Dr. Stanfield completed post-doctoral training at the University of Iowa and at the University of Alabama at Birmingham. She joined the faculty of the University of South Alabama in 1992.
Dr. Stanfield teaches Human Physiology 1 and Human Physiology 2, a Physiology Laboratory course, an On-line Physiology course, and Neuroscience.
Dr. Stanfield’s hobbies include
athletics, expecially softball, basketball and racquetball. She participates in intramural
sports with the AED and the BMD Society. Dr. Stanfield also enjoys vegetable gardening, bonsai plants,
and fossil hunting.
Information on
various health professions can be found at the following site:
HEALTH
PROFESSION ADVISING
Dr. Stanfield’s research
concerns the modulation of pain, with an emphasis on how visceral stimuli may alter the
perception of pain. Her current research is focusing on vagal afferents and their role in
activating descending pain modulatory systems in the rostral ventral medulla.
Thurston-Stanfield,C.L., Effects of temperature and volume on intraperitoneal saline-induced changes in blood pressure, nociception, and neural activity in the rostroventral medulla., Brain Research, 951 (2002) 59-68.
Thurston-Stanfield, C.L., Ranieri, J.T., Vallabhapurapu, R., and Barnes-Noble, D., Role of vagal afferents and the rostral ventral medulla in intravenous serotonin-induced changes in nociception and arterial blood pressure., Physiology & Behavior, 67 (1999) 753-767.
Thurston, C.L. and Helton, E.S., Effects of intravenous phenylephrine on blood pressure, nociception, and neural activity in the rostral ventral medulla in rats., Brain Research, 717 (1996) 81-90.
Thurston, C.L. and Randich, A., Responses of ON and OFF cells in the rostral ventral medulla to stimulation of vagal afferents and changes in mean arterial blood pressure in intact and cardiopulmonary deafferented rats., Pain, 62 (1995) 19-38.
Thurston, C.L., Starnes, A., and Randich, A., Changes in nociception, arterial blood pressure, and heart rate produced by intravenous morphine in the conscious rat., Brain Research, 612 (1993) 70-77.
Thurston, C.L., Randich, A., Electrical stimulation of the subdiaphragmatic vagus in rats: inhibition of heat-evoked responses of spinal dorsal horn neurons and central substrates mediating inhibition of the nociceptive tail flick reflex., Pain, 51 (1992) 349-365.
Randich, A., Thurston, C.L., Ludwig, P.S., Robertson, J.D., and Rasmussen, C., Intravenous morphine-induced activation of vagal afferents: Peripheral, spinal, and CNS substrates mediating inhibition of spinal nociception and cardiovascular responses., J. Neurophysiol., 68 (1992) 1027-1045.
Thurston, C.L., Campbell, I.G., Culhane, E.S., Carstens, E., and Watkins, L.R., Characterization of intrathecal vasopressin-induced antinociception, scratching behavior, and motor suppression., Peptides, 13 (1992) 17-25.
Thurston, C.L., and Randich, A., Effects of vagal afferent stimulation on ON and OFF cells in the rostroventral medulla: Relationships to nociception and arterial blood pressure., J. Neurophysiol., 67 (1992) 180-196.
Randich, A., and Thurston, C.L., Antinociceptive states and hypertension., J. Cardiovasc. Electrophysiol., 2 (1990) S54-S58.
Randich, A., Thurston, C.L., Ludwig, P.S., Timmerman, M.R., and Gebhart, G.F., Antinociception and cardiovascular responses produced by intravenous morphine: The role of vagal afferents., Brain Research, 543 (1990) 256-270.
Thurston, C.L., and Randich, A., Quantitative characterization and spinal substrates of antinociception produced by electrical stimulation of the subdiaphragmatic vagus in rats., Pain, 44 (1990) 201-210.
Watkins, L.R., Thurston, C.L., and Fleshner, M., Phenylephrine-induced antinociception: Investigations of potential neural and endocrine bases., Brain Research, 528 (1990) 273-284.
Thurston, C.L., and Randich, A., Acute increases in arterial blood pressure produced by occlusion of the abdominal aorta induces antinociception: Peripheral and central substrates., Brain Research, 519 (1990) 12-22.
Thurston, C.L., Culhane, E.S., Suberg, S.N., Carstens, E., and Watkins, L.R., Antinociception versus motor effects of intrathecal vasopressin as measured by four pain tests., Brain Research, 463 (1988) 1-11.
Watkins, L.R., Suberg, S.N., Thurston, C.L., and Culhane, E.S., Effects of spinal neuropeptides on pain sensitivity and analgesia: Thyrotropin releasing hormone and vasopressin., Brain Research, 362 (1986) 308-317.
UNIVERSITY OF
SOUTH ALABAMA
Department of Biomedical Sciences
Webmaster: Cindy L. Stanfield
Last updated: January 16, 2006
URL:
http://www.southalabama.edu/alliedhealth/biomedical