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NIH - Funded Training in Cell Signaling & Lung Pathobiology
 

Brad Swiger
Phone: (251) 460-6786
Fax: (251) 460-6798
bms304@jaguar1.usouthal.edu

   
EDUCATION

B.S. in Biomedical Sciences, University of South Alabama, 2001
Ph.D. in Basic Medical Sciences, University of South Alabama, expected 2008

   

MAJOR RESEARCH INTERESTS

Hypoxic Gene Regulation
Free Radical Biology
Mitochondrial Biology
Signaling Microdomains
Organelle Trafficking

 

SOCIETIES AND ORGANIZATIONS>

Alpha Theta Chi
American Association for the Advancement of Science
American Society for Pharmacology and Experimental Therapeutics
Basic Medical Sciences Student Organization

   

ADMINISTRATIVE EXPERIENCE

Basic Medical Sciences Student Organization Vice-President 2003-2004
Basic Medical Sciences Student Organization President 2004-2005
Graduate Program Executive Committee Student Representative 2004-2005
Science Olympiad, Event Judge 2005
Biomedical Enrichment and Recruitment Program (BEAR), Medical Cell Biology Course Director 2006

   

TEACHING EXPERIENCE

2003 Medical Pharmacology, Biomedical Enrichment and Recruitment Program (BEAR)
2004 Medical Pharmacology, BEAR
2004 Medical Cell Biology, BEAR
2005 Medical Cell Biology, BEAR
2006 Medical Cell Biology, BEAR
2007 Medical Cell Biology, BEAR
   

HONORS AND AWARDS

2003 Favorite Teacher Award, BEAR
2007 2nd place, contest for giving scientific presentations, course IDL 650
   

RESEARCH GRANTS

American Heart Association Pre-doctoral Fellowship, “Prolyl hydroxylase inhibition stimulates reactive oxygen species production in rat liver mitochondria”, 2004-2006, $36,000.
 

INVITED LECTURES

2004 Mechanisms in mitochondrial oxygen sensing, Cell Signaling Seminar Series, University of South Alabama
2004 Prolyl hydroxylase inhibition induces cytochrome c-dependent release of reactive oxygen species from isolated mitochondria:  a potential mechanism in oxygen sensing,  American Thoracic Society, Orlando, FL
 

PRESENTATIONS

2002 Research in Progress, Center for Lung Biology, University of South Alabama, November
2003 Research in Progress, Center for Lung Biology, University of South Alabama, March and September
2004 Journal Club, Mitochondrial Focus Group, University of South Alabama, October
2004 Research in Progress, Center for Lung Biology, University of South Alabama, November
2005 Journal Club, Mitochondrial Focus Group, University of South Alabama, May
2006 Research in Progress, Center for Lung Biology, University of South Alabama, April
2007 Research in Progress, Center for Lung Biology, University of South Alabama, March


ABSTRACTS

2004 Prolyl hydroxylase inhibition induces cytochrome c-dependent release of reactive oxygen species from isolated mitochondria: a potential mechanism in oxygen sensing, American Journal of Respiratory and Critical Care Medicine, 169(7): A512, 2004.
2006 Thrombin induces oxidative mitochondrial DNA damage through a membrane NADPH oxidase in pulmonary microvascular endothelial cells, FASEB Journal, 20: A669, 2006.
2007 Hypoxia causes mitochondrial translocation to the peri-nuclear region: implications for signal transduction, FASEB Journal, 21: 762.2, 2007.
   
   
  Last update: July 01, 2007